Update on CCR5 Delta 32 and death

The CCR5 Delta 32 Deletion has been shown to be a cause of death in influenza cases.

Evolutionists think this is a "good" or beneficial mutation.  How can something that is found to be a cause of suffering and death in over 20 different diseases, be considered "beneficial"?  Because humans seem to be without reasonableness and groping to validate many forms of compulsions, desires, greed, and ideology.
Here is the first two articles on this horrible mutation that causes suffering.  It really aggravates Evolutionists when they see that I use only peer reviewed medical science from all over the earth as my data.

http://evolutionsciencenow.blogspot.com/2013/06/ccr5-delta-32-deletion-mutation-hiv-and.html

http://evolutionsciencenow.blogspot.com/2014/09/more-on-ccr5-delta-32.html

CCR5 deficiency predisposes to fatal outcome in influenza virus infection

1. Ana Falcon, Dr. (afalcon@cnb.csic.es)1, 
2. Maria T Cuevas, Dr.2,
3. Ariel Rodriguez-Frandsen, Dr.3, 
4. Noelia Reyes4,
5. Francisco Pozo, Dr.5, 
6. Silvia Moreno6, 
7. Juan Ledesma, Dr.7,
8. Jose Martínez-Alarcón, Dr.8, 
9. Amelia Nieto, Dr.9 and
10. Inmaculada Casas, Dr.10

+Author Affiliations

1. 1 National Centre of Biotechnology (CNB-CSIC)
2. 2 Instituto de Salud Carlos III. Madrid, Spain
3. 3 National Center of Biotechnology (CNB-CSIC). Madrid, Spain
4. 4 Instituto de Salud Carlos III. Madrid, Spain
5. 5 Instituto de Salud Carlos III. Madrid, Spain
6. 6 Instituto de Salud Carlos III. Madrid, Spain
7. 7 Instituto de Salud Carlos III. Madrid, Spain
8. 8 Hospital General de Ciudad Real. Ciudad Real, Spain.
9. 9 National Centre of Biotechnology (CNB-CSIC). Madrid, Spain
10. 10 Instituto de Salud Carlos III. Madrid, Spain

• Received 10 March 2015.
• Revised 23 April 2015.
• Accepted 24 April 2015.

Abstract

Influenza epidemics affect all age groups, although children, the elderly, and those with underlying medical conditions are the most severely affected. Whereas co-morbidities are present in 50% of fatal cases, 25-50% of deaths are of apparently healthy individuals. This suggests underlying genetic determinants that govern infection severity. Although some viral factors that contribute to influenza disease are known, the role of host genetic factors remains undetermined. Data for small cohorts of influenza-infected patients are contradictory regarding the potential role of chemokine receptor 5 deficiency (CCR5-Δ32 mutation, a 32-base pair deletion in CCR5) in the outcome of influenza virus infection. We tested 171 respiratory samples from influenza patients (2009 pandemic) for CCR5-Δ32 and evaluated its correlation with patient mortality. CCR5-Δ32 patients (17.4%) showed a higher mortality rate than wild-type individuals (4.7%; p = 0.021), which indicates that CCR5-Δ32 patients are at higher risk than the normal population of fatal outcome in influenza infection.