Sunday, August 30, 2015

More Absolutely Irrefutable Evidence of Humanity's Condition: Author, James Arjuna

All of the evidence from the historical records of the rapid rise of diseases,



The historical records of massive new huge hospitals treating diseases that did not exist before the 1700's
The medical industry did not exist prior to the 1600's.  So from nearly ZERO costs to $4,200,000,000,000 (2015 figures) per year in the USA is an indication of the complete failure of humanity to curb their genetic suicide.  That is over $13,000  spent per citizen per year on medical and still there are no cures.  If you spent 43,000 years counting one dollar at a time, you would get to 4.2 TRILLION, just to give you some perspective.



Greed, lust, anger, division, hate, race, are all disgusting human characteristics.    Greed is a disease and it causes human diseases and suffering.  Lust is a disease and we can see how that works.   300% rise in Childhood cancer in 45 years!

Diseased (STD"s) parents have diseased babies.  It is not magical.  You cannot have rampant STD's and the highest rate of STD's in a country (USA) and not have diseased babies.   How can we continue to destroy all life on earth for greed?

It is not even stoppable by humans, because humans are the problem.  No morals, no science, and the medical industry is a total failure.

12,000,000 causes of medical misdiagnoses causing harm to patients.

http://www.livescience.com/44888-misdiagnosis-doctors-visits.html

The third leading cause of death is death by doctor.


*Deaths by medical mistakes hit records*
400,000 people die each year from medical mistakes.
http://www.healthcareitnews.com/news/deaths-by-medical-mistakes-hit-records

Number one cause of death  Heart disease.   A proven genetic disease in which the arteries are etched by the immune system and that normally slick artery becomes like a gravel road and it collects lipids.

Number 2 cause of Death is Cancer, proven by over 500,000 peer DNA studies to be a genetic disease of the immune system.  Cancer cells are a normal occurrence in humans.  The healthy immune system will destroy those defective cells. The weak and defective immune system cannot even recognize them as defective anymore.

Number 3 cause of death,  Medical incompetence.  The MD's today have all sorts of technology and still they cannot diagnoses and treat diseases correctly because they are retarded from mutations causing an overall retardation of humans.  Memory loss is the most common disease.

If the medical industry was a success there would be a reduction in diseases leading to no disease at all.  Why do people think that having more and more doctors, medical centers, and diseases is a good thing? 

http://evolutionsciencenow.blogspot.com/2013/06/the-medical-industry-and-what-it-tells.html

We cannot even build a simple jet airplane because it is continually grounded for safety issues. Now, the DC 3 aircraft made in the 1930's is still a functional airplane after 80 years.  http://www.cnn.com/2014/06/04/travel/aviation-douglas-dc-3/

https://en.wikipedia.org/wiki/Douglas_DC-3

We cannot make a rocket that will not explode, because of mental degradation in the engineers.

http://www.space.com/27583-antares-rocket-explosion-captured-by-journalist-video.html

In 1969 we put a man on the moon with a rudimentary mechanical calculator, and slide rules to make calculations on engineering.  I know because I was there.  The difference is the much higher intelligence of that generation.

In less that 4 generations, 1914, we went from less than 2% of high school students who were "special" (we called them retarded because that is a medical term for low intelligence).  Less than 2% of seniors in high school could not pass an English, Math, History, Science test in order to graduate.   (Now, that test would be useless, because less than 20% could pass it today.)

Now 40% of high school senior students are incapable of reading in English, writing in English nor can they do simple math.  They cannot even pass any college entrance exams and the universities have drastically lowered their academic standards, because if they did not do that there would be no "students".

http://nces.ed.gov/nationsreportcard/pubs/main2005/2007468.asp

Only 23% of students tested above BASIC standards. That would mean that 23% got a "D" or above.  In my day if you got a D you were considered "retarded", "lazy", and not wanting to learn.

We are watching a human tragedy take place with mental retardation as the normal human.   There are very few people who can concentrate long enough to even read my postings.   If you can read and understand this, you are in the top 10% of intelligence on the planet.

Humanity is degrading faster than ever.  Over ten times the diseases, over 20 times the mental retardation.

For over 200,000 years (by some unproven dating methods) humans did not write things down, because they had a nearly perfect memory.
Like all technology. The invention of writing is evidence of human degradation.

For over 200,000 years humans (by some unproven dating methods) lived with no medical, because disease was not prevalent.
Like all technology. The invention of medical treatments came from genetic degradation.

Now even with all the electronic memories we cannot keep from killing 400,000 humans a year from medical incompetence.

No wonder there are so many "believers" in the evolution fraud.

http://evolutionsciencenow.blogspot.com/2013/06/the-medical-industry-and-what-it-tells.html

Monday, August 24, 2015

What is the Truth about Humanity; The REAL SCIENCE on Humans; Author, James Arjuna


You see in real science we use evidence.    All the evidence shows only one thing as far as evolution.




When seeking the truth in the phenomenon all the evidence we can test and study always shows the same conclusion, or it is not science.  The truth is revealed when you remove your emotions, prejudices and the garbage beliefs from the study.  The Truth about humans always evokes strong emotions of denial in people, simply because most are guilty of supporting the cause of diseases from the condition of ignorance. And humans are emotional and it is very difficult to deal with reality.


The reason why the theroy of evolution cannot be proved is because they misinterpret all the data to say what the government wants to say.  There is no government controlled by the rich plutocrats going to allow the truth about how horribly they have managed life on earth.  The same people who sell you "save the planet", "save the whales", "save the children" and "save the chimpanzees" are the ones who are destroying all life on earth, human greed.


They are not concerned about you, your children, nor your grandchildren.  The nature of greed is total selfishness and concern for one thing MORE and MORE power and money.


So they make up magical reasons why humans are so sickly and die so young.   The medical industry is evidence of human failures.   The medical industry in the USA spends over (2021 states) $15,625 per citizen per year in all the medical related revenues being transferred.  Disease is a "good" business.   over $3,800,000,000,000 per year in just the USA.  Forbes magazine now quotes over (2015) 4.2 trillion dollars used in the medical industry. Update it is now over $5 TRILLION DOLLARS (2021)


Every genetic disease rising by 10 times per capita (per 100,000 people) in less than 100 years. Some are more like 15 times (diabetes).  And we have accumulated some 18,000  (2021) genetic diseases,  The cause is (99.9%) infected reproduction.   You cannot share any sexual contact outside of a monogamous uninfected (clean of all mutagenic pathogens:  viral, fungal, amoebic, bacterial STD's)  marriage and have healthy babies.  And once you have any of the standard viral STD's, they never go away.  It becomes part of your reproductive cells.  There is no cure for viruses.  They can remain "dormant" and held at bay by your immune system for 50 years, then blossom as soon as your immune system gets weaker. 


Cancer was 1 in 45 in 1847 as a cause of death.  Cancer was 1 in 20 at the start of the 20th century (1900).   Now it is 1 in 3 dying of this disease before the age of 64.
Congenital heart disease is up 200% in 29 years. We now have so many diseases that are brand new, never were seen before in all the historical records of humanity.
We now have huge cancer hospitals strictly for the treatment of children.  If we did not have so many sick children they would never exist.  These are all new in the last 15 to 20 years, because of the phenomenal rise of childhood cancers.

Death by Doctor is now number three killer in the USA.

12,000,000 causes of medical misdiagnoses causing harm to patients. 

http://www.livescience.com/44888-misdiagnosis-doctors-visits.html

The third leading cause of death is death by doctor.


*Deaths by medical mistakes hit records*
400,000 people die each year from medical mistakes.
http://www.healthcareitnews.com/news/deaths-by-medical-mistakes-hit-records

The CDC has issued Biblical instructions for stopping STD's.  This is because STD's have risen by 20,000,000 cases each year for the past 11 years.  Most of the infected are young girls ages 13 to 24.  Second is males 13 to 24. (All at peak reproductive years) These "kids" are just  following the sexual misconduct as being good ideas of society.   Movies with sex, violence and killing as the constant and well received theme by stupid people.  Promoting sexual misconduct as being good in the classroom.  It is pure irony that most of the movie "stars" all support gun control and taking guns away from citizens, and they use guns in nearly every movie they make, brainwashing young people into a violent mindset.  They also have sex for no reason with random sexual partners as if that was a good thing to do. Now many of the famous actors, and actresses are diseased with HPV, HSV-1, HSV-1, HIV/AIDS and Hepatitis is rampant in that industry. These are the "gods" that young people honor?  We are screwed as a society.


The CDC clearly states that there is no safe sex outside of a marriage to an uninfected person.  (You cannot find an uninfected person according to STD statistics)This is because this massive sales job on condom use was a lie.  Kissing, touching, oral contact with any sexual area infected is not  affected by condom use.  Skin to skin contact around the "condom" will give you several diseases.   You cannot deep kiss and not get infected with HSV-1, HSV-2, HPV, HBV. HIV (HIV; if there is any  blood contact, and this is more common than you think).

It is really far worse than people "want to believe". And when your baby is born with some disease that did not come from any inheritance of diseases, it came from infected reproduction.  Viral infections at conception destroy the original coding of the DNA by reverse transcriptase into the cells.  And if this is the ONLY cell you have then all the other cells will be screwed up also.


Humans are totally infected today and will never allow any idea of saving the lives of the offspring to enter into the compulsive sexual immorality.   Sex is for reproduction with a clean uninfected mate.  You can have all the sex you want with ONE person but if you go outside the odds are extreme that you will pick up a disease now and that disease is permanent.


This is why we are doomed as a species to suffering, diseases, continual rise of retardation, and mothers taking their children to the hospital over and over trying to keep them alive from some horrible disease.  I have studied many hundreds of cases to the point of reading mothers "blogs" on the torture of taking their precious beautiful babies to the hospital as many as 50 times to keep them alive, only to have them die after the 10th surgery.  These new diseases are extremely horrible.


This is what the evidence shows.

Thursday, June 11, 2015

CCR5 Delta 32 Deletion more diseases caused by this mutation.





  1. Akcay A, Nguyen Q, Edelstein CL (2009) Mediators of inflammation in acute kidney injury. Mediat Inflamm 2009:137072. doi:10.​1155/​2009/​137072 View Article
  2. Albelda SM, Smith CW, Ward PA (1994) Adhesion molecules and inflammatory injury. FASEB J 8(8):504–512PubMed
  3. Anders HJ, Vielhauer V, Frink M et al (2002) A chemokine receptor CCR-1 antagonist reduces renal fibrosis after unilateral ureter ligation. J Clin Invest 109(2):251–259. doi:10.​1172/​JCI14040 PubMed CentralPubMedView Article
  4. Barcellos LF, Schito AM, Rimmler JB et al (2000) CC-chemokine receptor 5 polymorphism and age of onset in familial multiple sclerosis. Multiple sclerosis genetics group. Immunogenetics 51(4–5):281–288PubMedView Article
  5. Broide DH, Humber D, Sullivan S, Sriramarao P (1998) Inhibition of eosinophil rolling and recruitment in P-selectin- and intracellular adhesion molecule-1-deficient mice. Blood 91(8):2847–2856PubMed
  6. Coenen M, Nattermann J (2010) The role of CCR5 in HCV infection. Eur J Med Res 15(3):97–101PubMed CentralPubMedView Article
  7. Cunningham PN, Dyanov HM, Park P, Wang J, Newell KA, Quigg RJ (2002) Acute renal failure in endotoxemia is caused by TNF acting directly on TNF receptor-1 in kidney. J Immunol 168(11):5817–5823PubMedView Article
  8. Cunningham PN, Wang Y, Guo R, He G, Quigg RJ (2004) Role of Toll-like receptor 4 in endotoxin-induced acute renal failure. J Immunol 172(4):2629–2635PubMedView Article
  9. Dawson TC, Beck MA, Kuziel WA, Henderson F, Maeda N (2000) Contrasting effects of CCR5 and CCR2 deficiency in the pulmonary inflammatory response to influenza A virus. Am J Pathol 156(6):1951–1959. doi:10.​1016/​S0002-9440(10)65068-7 PubMed CentralPubMedView Article
  10. Day YJ, Huang L, Ye H, Linden J, Okusa MD (2005) Renal ischemia-reperfusion injury and adenosine 2A receptor-mediated tissue protection: role of macrophages. Am J Physiol Renal Physiol 288(4):F722–F731. doi:10.​1152/​ajprenal.​00378.​2004 PubMedView Article
  11. Dong Z, Atherton SS (2007) Tumor necrosis factor-alpha in cisplatin nephrotoxicity: a homebred foe? Kidney Int 72(1):5–7. doi:10.​1038/​sj.​ki.​5002320 PubMedView Article
  12. Fadel SA, Bromley SK, Medoff BD, Luster AD (2008) CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality. Eur J Immunol 38(12):3376–3387. doi:10.​1002/​eji.​200838628 PubMed CentralPubMedView Article
  13. Fantuzzi G, Zheng H, Faggioni R et al (1996) Effect of endotoxin in IL-1 beta-deficient mice. J Immunol 157(1):291–296PubMed
  14. Fattori E, Della Rocca C, Costa P et al (1994) Development of progressive kidney damage and myeloma kidney in interleukin-6 transgenic mice. Blood 83(9):2570–2579PubMed
  15. Faubel S, Ljubanovic D, Reznikov L, Somerset H, Dinarello CA, Edelstein CL (2004) Caspase-1-deficient mice are protected against cisplatin-induced apoptosis and acute tubular necrosis. Kidney Int 66(6):2202–2213. doi:10.​1111/​j.​1523-1755.​2004.​66010.​x PubMedView Article
  16. Furuichi K, Wada T, Iwata Y et al (2003) CCR2 signaling contributes to ischemia-reperfusion injury in kidney. J Am Soc Nephrol 14(10):2503–2515PubMedView Article
  17. Furuichi K, Gao JL, Horuk R, Wada T, Kaneko S, Murphy PM (2008) Chemokine receptor CCR1 regulates inflammatory cell infiltration after renal ischemia-reperfusion injury. J Immunol 181(12):8670–8676PubMed CentralPubMedView Article
  18. Gadient RA, Patterson PH (1999) Leukemia inhibitory factor, Interleukin 6, and other cytokines using the GP130 transducing receptor: roles in inflammation and injury. Stem Cells 17(3):127–137. doi:10.​1002/​stem.​170127PubMedView Article
  19. Homsi E, Ribeiro-Alves MA, Lopes de Faria JB, Dias EP (2002) Interleukin-6 stimulates tubular regeneration in rats with glycerol-induced acute renal failure. Nephron 92(1):192–199PubMedView Article
  20. Jo SK, Cho WY, Sung SA, Kim HK, Won NH (2005) MEK inhibitor, U0126, attenuates cisplatin-induced renal injury by decreasing inflammation and apoptosis. Kidney Int 67(2):458–466. doi:10.​1111/​j.​1523-1755.​2005.​67102.​x PubMedView Article
  21. Kayama F, Yoshida T, Elwell MR, Luster MI (1995) Cadmium-induced renal damage and proinflammatory cytokines: possible role of IL-6 in tubular epithelial cell regeneration. Toxicol Appl Pharmacol 134(1):26–34. doi:10.​1006/​taap.​1995.​1165 PubMedView Article
  22. Kelly KJ, Williams WW Jr, Colvin RB, Bonventre JV (1994) Antibody to intercellular adhesion molecule 1 protects the kidney against ischemic injury. Proc Natl Acad Sci U S A 91(2):812–816PubMed CentralPubMedView Article
  23. Knotek M, Rogachev B, Wang W et al (2001) Endotoxemic renal failure in mice: role of tumor necrosis factor independent of inducible nitric oxide synthase. Kidney Int 59(6):2243–2249. doi:10.​1046/​j.​1523-1755.​2001.​00740.​xPubMedView Article
  24. Lee YK, Kwak DH, Oh KW et al (2009) CCR5 deficiency induces astrocyte activation, Abeta deposit and impaired memory function. Neurobiol Learn Mem 92(3):356–363. doi:10.​1016/​j.​nlm.​2009.​04.​003 PubMedView Article
  25. Li L, Huang L, Sung SS et al (2007) NKT cell activation mediates neutrophil IFN-gamma production and renal ischemia-reperfusion injury. J Immunol 178(9):5899–5911PubMedView Article
  26. Linas SL, Shanley PF, Whittenburg D, Berger E, Repine JE (1988) Neutrophils accentuate ischemia-reperfusion injury in isolated perfused rat kidneys. Am J Physiol 255(4 Pt 2):F728–F735PubMed
  27. Mehta RL, Kellum JA, Shah SV et al (2007) Acute kidney injury network: report of an initiative to improve outcomes in acute kidney injury. Crit Care 11(2):R31. doi:10.​1186/​cc5713 PubMed CentralPubMedView Article
  28. Meldrum KK, Meldrum DR, Hile KL et al (2001) p38 MAPK mediates renal tubular cell TNF-alpha production and TNF-alpha-dependent apoptosis during simulated ischemia. Am J Physiol Cell Physiol 281(2):C563–C570PubMed
  29. Messmer UK, Briner VA, Pfeilschifter J (1999) Tumor necrosis factor-alpha and lipopolysaccharide induce apoptotic cell death in bovine glomerular endothelial cells. Kidney Int 55(6):2322–2337. doi:10.​1046/​j.​1523-1755.​1999.​00473.​xPubMedView Article
  30. Mishima K, Baba A, Matsuo M, Itoh Y, Oishi R (2006) Protective effect of cyclic AMP against cisplatin-induced nephrotoxicity. Free Radic Biol Med 40(9):1564–1577. doi:10.​1016/​j.​freeradbiomed.​2005.​12.​025 PubMedView Article
  31. Moreno C, Gustot T, Nicaise C et al (2005) CCR5 deficiency exacerbates T-cell-mediated hepatitis in mice. Hepatology 42(4):854–862. doi:10.​1002/​hep.​20865 PubMedView Article
  32. Patel NS, Chatterjee PK, Di Paola R et al (2005) Endogenous interleukin-6 enhances the renal injury, dysfunction, and inflammation caused by ischemia/reperfusion. J Pharmacol Exp Ther 312(3):1170–1178. doi:10.​1124/​jpet.​104.​078659PubMedView Article
  33. Perez de Lema G, Maier H, Franz TJ et al (2005) Chemokine receptor Ccr2 deficiency reduces renal disease and prolongs survival in MRL/lpr lupus-prone mice. J Am Soc Nephrol 16(12):3592–3601. doi:10.​1681/​ASN.​2005040426PubMedView Article
  34. Ramesh G, Reeves WB (2002) TNF-alpha mediates chemokine and cytokine expression and renal injury in cisplatin nephrotoxicity. J Clin Invest 110(6):835–842. doi:10.​1172/​JCI15606 PubMed CentralPubMedView Article
  35. Ramesh G, Reeves WB (2003) TNFR2-mediated apoptosis and necrosis in cisplatin-induced acute renal failure. Am J Physiol Renal Physiol 285(4):F610–F618. doi:10.​1152/​ajprenal.​00101.​2003 PubMedView Article
  36. Ramesh G, Reeves WB (2005) p38 MAP kinase inhibition ameliorates cisplatin nephrotoxicity in mice. Am J Physiol Renal Physiol 289(1):F166–F174. doi:10.​1152/​ajprenal.​00401.​2004 PubMedView Article
  37. Ramesh G, Kimball SR, Jefferson LS, Reeves WB (2007) Endotoxin and cisplatin synergistically stimulate TNF-alpha production by renal epithelial cells. Am J Physiol Renal Physiol 292(2):F812–F819. doi:10.​1152/​ajprenal.​00277.​2006PubMedView Article
  38. Remick DG, Newcomb DE, Bolgos GL, Call DR (2000) Comparison of the mortality and inflammatory response of two models of sepsis: lipopolysaccharide vs. cecal ligation and puncture. Shock 13(2):110–116PubMedView Article
  39. Russell JA, Singer J, Bernard GR et al (2000) Changing pattern of organ dysfunction in early human sepsis is related to mortality. Crit Care Med 28(10):3405–3411PubMedView Article
  40. Rybak SL, Murphy RF (1998) Primary cell cultures from murine kidney and heart differ in endosomal pH. J Cell Physiol 176(1):216–222. doi:10.​1002/​(SICI)1097-4652(199807)176:​1<216:​AID-JCP23>3.​0.​CO PubMedView Article
  41. Samson M, Labbe O, Mollereau C, Vassart G, Parmentier M (1996) Molecular cloning and functional expression of a new human CC-chemokine receptor gene. Biochemistry 35(11):3362–3367. doi:10.​1021/​bi952950g PubMedView Article
  42. Schrier RW, Wang W (2004) Acute renal failure and sepsis. N Engl J Med 351(2):159–169. doi:10.​1056/​NEJMra032401351/​2/​159 PubMedView Article
  43. Segerer S, Nelson PJ, Schlondorff D (2000) Chemokines, chemokine receptors, and renal disease: from basic science to pathophysiologic and therapeutic studies. J Am Soc Nephrol 11(1):152–176PubMed
  44. Segerer S, Banas B, Wornle M et al (2004) CXCR3 is involved in tubulointerstitial injury in human glomerulonephritis. Am J Pathol 164(2):635–649. doi:10.​1016/​S0002-9440(10)63152-5 PubMed CentralPubMedView Article
  45. Sorce S, Bonnefont J, Julien S et al (2010) Increased brain damage after ischaemic stroke in mice lacking the chemokine receptor CCR5. Br J Pharmacol 160(2):311–321. doi:10.​1111/​j.​1476-5381.​2010.​00697.​x PubMed CentralPubMedView Article
  46. Turner JE, Paust HJ, Steinmetz OM et al (2008) CCR5 deficiency aggravates crescentic glomerulonephritis in mice. J Immunol 181(9):6546–6556PubMedView Article
  47. Turner JE, Paust HJ, Bennstein SB et al (2012) Protective role for CCR5 in murine lupus nephritis. Am J Physiol Renal Physiol 302(11):F1503–F1515. doi:10.​1152/​ajprenal.​00382.​2011 PubMedView Article
  48. Wang W, Faubel S, Ljubanovic D et al (2005) Endotoxemic acute renal failure is attenuated in caspase-1-deficient mice. Am J Physiol Renal Physiol 288(5):F997–F1004. doi:10.​1152/​ajprenal.​00130.​2004 PubMedView Article
  49. Xu C, Chang A, Hack BK, Eadon MT, Alper SL, Cunningham PN (2014) TNF-mediated damage to glomerular endothelium is an important determinant of acute kidney injury in sepsis. Kidney Int 85(1):72–81. doi:10.​1038/​ki.​2013.​286 PubMedView Article
  50. Yanaba K, Mukaida N, Matsushima K, Murphy PM, Takehara K, Sato S (2004) Role of C-C chemokine receptors 1 and 5 and CCL3/macrophage inflammatory protein-1alpha in the cutaneous Arthus reaction: possible attenuation of their inhibitory effects by compensatory chemokine production. Eur J Immunol 34(12):3553–3561. doi:10.​1002/​eji.​200425426 PubMedView Article
  51. Zhang D, Li Y, Liu Y, Xiang X, Dong Z (2013) Paclitaxel ameliorates lipopolysaccharide-induced kidney injury by binding myeloid differentiation protein-2 to block Toll-like receptor 4-mediated nuclear factor-kappaB activation and cytokine production. J Pharmacol Exp Ther 345(1):69–75. doi:10.​1124/​jpet.​112.​202481 PubMed CentralPubMedView Article